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Auditory stimuli as a trigger for arrhythmic events differentiate HERG-related (LQTS2) patients from KVLQT1-related patients (LQTS1).

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Mice with cardiac-specific overexpression of a dominant negative mutant of AMPK have depressed rates of glucose uptake (), suggesting a critical role for AMPK in regulating basal glucose uptake in the heart.

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These conditions contribute to increased risk for cardiovascular disease (NDIC, 2014).

The weekly sublingual nitrate use was also lower in the treatment group (1.

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Heparin administration requires careful monitoring for bleeding, and when the drug is stopped, the patient must be watched for “rebound” ischemic episodes that sometimes occur during the subsequent 24 hours.

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More importantly, treatment with the other anti-anginal drugs did not significantly modify the response to ranolazine.

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Specific therapy for primary causes of ischemia should be directed at each pathophysiologic origin of unstable angina: increased myocardial rate-pressure product, coronary vasoconstriction, platelet aggregation, and thrombosis.

Lopaschuk is a Medical Scientist of the Alberta Heritage Foundation for Medical Research and was supported by the Canadian Institutes of Health.

Exercise stress tests were performed at the end of each treatment period.

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The chi-square test was used to compare the proportions of patients in each treatment group who were angina-free as defined by the angina-frequency score on the Seattle Angina Questionnaire (with a score of 100 indicating that the patient was angina-free).

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